Lidocaine Hydrochloride Powder Lidocaina Hcl 99% Pure Safe Clearance EP Standard
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Lidocaina hcl Basic Information
What is Lidocaina Hcl ?
Lidocaina Hcl Physical and chemical properties
White crystal, odorless, slightly bitter and numb. Very soluble in water, ethanol and organic solvents, but insoluble in ether. The aqueous solution does not decompose in the case of acid and alkali, and it rarely deteriorates after repeated autoclaving.
Lidocaina Hcl is kind of Local anesthetic
Lidocaina Hcl is kind of Antiarrhythmic Drugs
Lidocaina Hcl can be used for Infiltration anesthesia
Lidocaina Hcl can be used for Epidural anesthesia,
Lidocaina Hcl can be used for Surface anesthesia (including mucosal anesthesia during thoracoscopy or abdominal surgery).
Lidocaina Hcl can be used for Nerve block
Lidocaina Hcl can be used for Premature ventricular contractions and ventricular tachycardia after acute myocardial infarction
Lidocaina Hcl can be used for Ventricular arrhythmia caused by digitalis poisoning.
Lidocaina Hcl can be used for Ventricular arrhythmias caused by cardiac surgery and cardiac catheterization.
Lidocaina hydrochloride is an amide-type local anesthetic.
After blood absorption or intravenous administration, it has an obvious biphasic effect of excitation and inhibition on the central nervous system, without pioneer excitation. When the plasma concentration is low, the threshold of analgesia, drowsiness and pain will increase; with the increase of the dose, the effect or toxicity will increase, and the anticonvulsant effect will be achieved when the plasma concentration is higher than the sub toxic plasma concentration; when the plasma concentration is higher than 5 μ g · ml-1, severe chemical book may occur. At low dose, lidocaine hydrochloride can promote K + outflow of cardiac myocytes, reduce cardiac autonomic ability, and have anti ventricular arrhythmia effect; at treatment dose, it affects electrical activity, and there is no obvious effect on atrioventricular conduction and contraction; further increase of blood concentration can lead to slow down of cardiac conduction velocity, atrioventricular conduction block, inhibition of cardiac contractility, and reduction of cardiac output.
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